14,15 The analysis of A. adeninivorans genome revealed a presence of almost all necessary genes essential for purine degradation to CO 2 and NH 3 (Fig. Disorders that involve abnormalities of nucleotide metabolism range from relatively common diseases such as hyperuricemia and gout, in which there is increased production or impaired excretion of a metabolic end product of purine metabolism (uric acid), to rare enzyme deficiencies that affect purine and pyrimidine synthesis or degradation. The end product of purine metabolism in primates including Dalmatian dog is uric acid. The end products of purine catabolism in various animals differ from those found in plants. In humans, most higher primates and birds, the urate oxidase gene is non-functional and hence uric acid is not further broken down. Uric acid (or urate) is a heterocyclic compound of carbon, nitrogen, oxygen, and hydrogen with the formula C5H4N4O3.. URIC ACID FORMATION In humans uric acid is the final oxidation (breakdown) product of purine metabolism. What is the percent dissociation of uric acid in urine at a typical pH = 6.0? By I. Russel Lee (129264), Liting Yang ...
While degradation of purines to uric acid is generally conserved among organisms, the end product of uric acid catabolism varies among taxa often due to the loss of functional catabolic enzymes in the pathway.
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